Protein essential for wound healing

A recent study conducted by researchers at the University of Washington School of Medicine could have significant implications for treating diseases linked to abnormal blood vessel growth, such as impaired wound healing in diabetes and vision loss due to macular degeneration. When an organ is injured, the body initiates a healing process that includes the formation of new blood vessels. Since vascular endothelial cells and blood cells play a key role in this process, the team led by Ornitz aimed to investigate what happens when they block the signals from FGFR1 and FGFR2, two major FGF signaling mediators found in these cells. Unlike previous studies, their approach involved more widespread inhibition of these signals. Surprisingly, the mice in the study appeared completely normal—running around and living long lives. However, when the researchers tested them, they observed slower wound healing and a significant reduction in blood vessel density around the injury site. This discovery highlights the critical role these proteins play in the healing process. The team also examined the eyes of the mice, where new blood vessels form in response to disease or damage. Unlike in other parts of the body, however, these new vessels in the eye are often problematic—they tend to bleed, leading to scarring and vision loss. The study suggests that increasing FGF signaling can enhance wound healing by promoting the growth of new blood vessels, especially in cases like diabetic foot ulcers. In fact, FGF2 has already been used in Japan as a topical treatment for such wounds. On the flip side, suppressing these pathways in the eye might contribute to conditions like age-related macular degeneration or diabetic retinopathy. In these cases, the body tries to compensate by forming new blood vessels, but these often worsen the condition instead of helping. The good news is that since these FGF pathways aren't involved in normal development or tissue maintenance, treatments that either boost or suppress them may not harm healthy tissues. In diabetes, retinal blood vessels weaken over time, and there's always concern that any therapy might damage these vessels. But the study shows that inhibiting FGF signaling in the eye can prevent the harmful overgrowth of blood vessels without affecting the healthy ones. This finding offers hope for more targeted and safer treatments for patients suffering from vision-threatening diseases.

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